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Current evidence suggests that the efficacy of SA in treating brain diseases, particularly ischemic strokes, is primarily achieved by stimulating the scalp nerves, especially the trigeminal nerve, to improve CBF. While the mechanisms of SA in treating various brain diseases may be multifaceted, understanding the neural regulation of SA on CBF captures the main aspects of its therapeutic mechanisms. This understanding also facilitates the elucidation of other mechanisms, which may be of greater significance for further clinical applications.